"Besides age and sex, the algorithm is based on simple routine data that is either available to therapists or can be collected easily, such as BMI, total cholesterol, or HbA1c. In this way, people who produce less insulin or tend to exhibit insufficiently controlled "Quote
from above "Both my Half-brother (same mother) and I became diabetic at age 45 with acute forms of ketoacidosis. My brother was deemed LADA Type 1, I was deemed Type 2
My story is one of total ignorance by clinicians, CDE, and Path Labs on HbA1c and unreliability due to ANY form of Anemia. Over a 20-year period, A1cs of 3.8% to 4.4% were considered "great Control"??? this explains how "silent hemoglobinopathies " confound A1c. Iron, Vit B12; and Folate deficiencies (common in Women especially) and, in my case, low-grade hemolytic anemia (acquired through chemicals used in engineering)
HbA1c should NEVER be used in isolation A fasting Glucose Reading, should always accompany an HbA1c. Pathology, NOT GPs, should compare these and alert GPs if some form of anemia is present. I have a fructosamine test. since discovering these blunders,
In Australia we use HbA1c <6.5% no diabetes as the Diagnostic test?? which allows our C.A.L.D populations susceptible to carrier traits of Sickle cell and Thalassemia, BAME populations in the UK, and Women in General, to fall through. the net of" Not being Diagnosed or Managed Medication wise"